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[Moreover, inhibition of p53 in PV erythroblasts resulted in more gamma-H2Ax (?-H2Ax)-marked double-stranded breaks compared with in like-treated ET erythroblasts, suggesting the defective intra-S checkpoint function seen in PV increases DNA damage in the context of attenuated p53 signaling.]. Sentence from MEDLINE/PubMed, a database of the U.S. National Library of Medicine.
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